This comprehensive review examines how metal dysregulation triggers specialized cell death pathways (ferroptosis, cuproptosis, zincoptosis, and others), with direct therapeutic applications in cancer and neurodegenerative disease. The article details both synthetic lethal strategies to eliminate therapy-resistant cancers and cytoprotective approaches to prevent pathological tissue damage. GHK-Cu operates within this copper-mediated mechanism framework, positioning it as a targeted metallobiological tool that practitioners should understand for both regenerative and potential anti-cancer applications.
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GHK-Cu fits within copper-mediated cell death pathways, informing its role in tissue regeneration and cellular stress modulation.